Scientific clues into eczema treatment


One major reason that I got into natural skincare hobby and started this blog is my son.  My son has eczema since he was a chubby 2-month old baby.  My child-caring experience is filled with searches for information on treating eczema, struggles for controlling eczema flare-ups, and sleepless long nights sitting next to my son’s bed and trying to calm him down so he can sleep.  I subscribe to science news feed and receive sciences news every day.  I am often excited to see any scientific progress made in eczema research, which I am sharing with you here.

Stopping the Itch: New Clues Into How to Treat Eczema


A recent research by Dr. Raif Geha, of Boston Children's Hospital, and his collaborators, showed that scratching the skin precipitates the condition by encouraging an influx of immune cells, neutrophils. These neutrophils secrete a lipid called leukotriene B4 that calls in more neutrophils, and more importantly, potent immune T cells that are the hallmark of eczema. These cells cause inflammation that aggravates the skin further. The investigators suspected that blocking the onslaught of these cells might slow down the disease or even stop it in its tracks. 

Furthermore, Dr. Geha and his colleagues wondered whether the production of leukotriene B4 served to recruit T cells to the site of mechanical insult. And indeed that was the case. "We showed that a drug that blocks the production of leukotriene B4 blocks the development of allergic skin inflammation in a mouse model of eczema," says Dr. Geha. His team also found that deleting the receptors on immune cells that bind to leukotriene B4 had a similar effect.

 

Lower Levels of Sunlight Exposure Link to Allergy and Eczema in Children, Study Suggests


Researchers from the European Centre for Environment & Human Health, along with several Australian institutions, have found that children living in areas with lower levels of sunlight are at greater risk of developing food allergies and the skin condition eczema, compared to those in areas with higher UV.

The report builds upon existing evidence that suggests exposure to the sun may play a role in rising levels of food allergy and eczema. Sunlight is important because it provides our body with the fuel to create vitamin D in the skin.

Prenatal Exposure to Common Household Chemical, Phthalate, Increases Risk for Childhood Eczema


Prenatal exposure to a ubiquitous household chemical called butylbenzyl phthalate (BBzP) can increase a child's risk for developing eczema, according to research conducted at the Columbia Center for Children's Environmental Health at the Mailman School of Public Health.

Researchers Discover Genetic Basis for Eczema, New Avenue to Therapies


Pharmaceutical scientists at OSU found in laboratory studies that eczema can be triggered by inadequate Ctip2, a protein and master regulator that affects other genetic functions. They have identified two ways in which improper function of Ctip2 can lead to eczema.

In a recent publication, they found that Ctip2 controls lipid biosynthesis in the skin, the fats that are needed to help keep skin healthy and hydrated. In the new study, they discovered that Ctip2 suppresses TSLP, a cytokine protein produced by skin cells that can trigger inflammation.

Levels of this inflammatory TSLP, which is ordinarily undetectable in human skin, were found to be 1,000 times higher in laboratory animals that had been genetically modified to have no Ctip2 production in their skin.

Protective Role of Skin Microbiota Described


A research team at the National Institutes of Health has found that bacteria that normally live in the skin may help protect the body from infection. These findings indicate that microbiota found in different tissues -- skin, gut, lung -- have unique roles at each site and that maintaining good health requires the presence of several different sets of commensal communities. This study provides new insights into the protective role of skin commensals and demonstrates that skin health relies on the interaction of commensals and immune cells.

Thanks for reading.

Connie


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